10.1016/0006-8993(89)90597-0 Brain Res
Oxidative phosphorylation, not glycolysis, powers presynaptic and postsynaptic mechanisms underlying brain information processing
In addition, in PD, the increased activity of NOx in microglia, the decreased activity of glutathione peroxidase in oligodendrocytes, the decreased activity of SOD in astrocytes, and the abnormal metabolism of glutathione all lead to the accumulation of intracellular superoxide, which in turn increases the occurrence of lipid peroxidation
In rodents, BPC-157 accelerates healing in alkali-burn wound models, coinciding with increased ERK1/2 activity in regenerating tissues, suggesting that this signaling axis contributes not only to cellular dynamics in vitro but also to tissue-level repair responses in vivo [26, 55]
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